New research reveals that bystander cell accumulation antagonizes rather than abets cell-killing by specific CTLs, curbing inflammation. The finding is noteworthy because that chief anti-inflammatory role has been traditionally ascribed to what are called regulatory T cells, or 'Tregs,' which dampen autoimmune responses. The new study challenges this notion and suggests alternate mechanisms can also be at work.
New research reveals that bystander cell accumulation antagonizes rather than abets cell-killing by specific CTLs, curbing inflammation. The finding is noteworthy because that chief anti-inflammatory role has been traditionally ascribed to what are called regulatory T cells, or 'Tregs,' which dampen autoimmune responses. The new study challenges this notion and suggests alternate mechanisms can also be at work.
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